Rapid Review·Cardiovascular

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Arrhythmias: Ventricular & Arrest

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Ventricular Tachycardia

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Key takeaways

three or more consecutive premature ventricular complexes from below the bundle of His, at 120 to 250 per minute.
non-sustained (under 30 seconds) or sustained (30 seconds or more).
monomorphic (uniform QRS) or polymorphic (varying QRS, as in Torsades de Pointes).
ischemic heart disease, non-ischemic cardiomyopathy and structural heart disease.
potassium or magnesium abnormalities, hypoxemia or acute ischemia, high sympathetic tone (sepsis).
often asymptomatic; otherwise palpitations, chest pain, dyspnea, dizziness, syncope, hypotension, or cardiac arrest.
wide QRS (over 120 ms) with signs of atrioventricular (AV) dissociation, as in the Monomorphic VT ECG.
  • AV dissociation: an atrial rate different from the ventricular rate, dissociated P waves, fusion and capture beats.
three or more consecutive wide complexes at 100 or more with AV dissociation.
search for and correct the cause (electrolytes); in a patient with no known predisposing disease, look for one (echocardiography).
immediate defibrillation under the cardiac arrest algorithm (Cardiac Arrest & ACLS).
intravenous amiodarone (lidocaine, procainamide or sotalol are alternatives).
synchronized cardioversion.
treated like pulseless VT, with defibrillation.
for patients at risk of recurrent VT or sudden death; beta blockers or amiodarone to suppress episodes.
three or more episodes of sustained VT, ventricular fibrillation or appropriate ICD shocks in 24 hours.
  • Most often after a myocardial infarction, in structural heart disease or an inherited arrhythmia.
  • Usually an indication for amiodarone.

How it's tested

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High-yield images2
A regular wide-complex tachycardia with uniform QRS morphology and independent P-wave activity is monomorphic ventricular tachycardia.
Non-sustained monomorphic VT: a run of 3 or more regular wide complexes over 100 per minute that stops on its own within 30 seconds. There is usually an underlying condition (cardiomyopathy, ischemia) or a trigger (hypokalemia, high sympathetic drive).

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# What is the best initial management for a hemodynamically stable patient with palpitations and the ECG findings below? 

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